Terbutaline

This article covers many, but not all, of the possible side effects with terbutaline.
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Seeing and being seen is critical to establishing connections. Attending key conferences is part of that process. Conferences should be work related and centered around your projects and technology. Your company should be willing to pay for these events. Attending at least one per year is a must. Autodesk University is the best startJ u l A. The amount of receptors bound at the ec 50 -value of clenbuterol was 8% compared to less than 1% for salbutamol and terbutaline.

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Probably at least four classes of receptors exist for catecholamines. Ahlquist I ; originally divided the effects of catecholamines into two groups which he called alpha and beta. Subsequently it was found that the beta effects of catecholamines are generally associated with increases in cyclic AMP 2 ; . Lands et al. 3 ; subdivided the beta effects of catecholamines into beta, and betap based on structure-activity relationships for agonists. The beta: ! adrenoceptors are involved in bronchodilation and are preferentially activated by epinephrine, while norepinephrine has little activity. In contrast, norepinephrine and epinephrine are equipotent activators of betal adrenoceptors in heart and adipose tissue 4 ; . Compounds with bulky groups tertiary butyl ; on the amine carbon such as terbutaline and albuterol are preferential beta2 agonists, while a compound such as prenalterol is more active at betal sites 4 ; . Ariens et al. 5 ; suggested that the betal effects are due to norepinephrine released by sympathetic nerves, while beta2 effects are due to hormonal effects of epinephrine released by the adrenal medulla Fig. 1 ; . Alpha effects of catecholamines are also divided into alpha, and alpha2 types 6-9 ; . There appear to be s arate receptors for the alphal versus the alpha2 effects 9 ; , which is hardly surprising since they have different biochemical actions. Wikberg 7 ; and Fain and GarciaSAinz 8 ; suggested that alphal effects involve turnover of phosphatidylinositol and elevation of cytosol Ca2 + , while alpha2 effects involve inhibition of adenylate cyclase. However, in cells in which the level of cyclic AMP affects the cytosol Cap + pool or the biochemical actions of Ca2 + , there will be effects of alpha2activation on Ca2 + action Table 1 ; . These cells are subject to what might best be called antagonistic control since Cap + and cyclic AMP have opposite effects 10 ; . Smooth muscle is a and baclofen. Sodium chloride soln, 22 sodium phosphate sodium biphosphate, 17 SOMA, 12 somatropin, 15 sotalol, 9 spacer device, 22 spironolactone, 10 spironolactone hydrochlorothiazide, 10 SPORANOX, 5 starch packets, 19 stavudine, 6 succimer, 16 sucralfate, 17 SUDAFED, 21 sulfacetamide 10%, 24 sulfacetamide prednisolone phosphate 10% 0.25%, 24 sulfamethoxazole trimethoprim, 6 sulfasalazine, 17 sulfasalazine delayed-rel, 17 sulindac, 3 sumatriptan, 12 SUMYCIN, 5 SUSTIVA, 5 SYNTHROID, 16 tacrine, 11 tacrolimus, 19, 23 TAGAMET, 16 TAMBOCOR, 9 TAMIFLU, 6 tamoxifen, 7 TAPAZOLE, 15 TARGRETIN, 7 tazarotene, 23 TAZORAC, 23 TEGRETOL, 10 TEGRETOL-XR, 10 temazepam, 11 TEMODAR, 7 TEMOVATE, 23 temozolomide, 7 TENEX, 8 TENORMIN, 9 terazosin, 8 terbinafine, 5 terbutaline, 21 TESSALON, 20 tetracycline, 5 THEOCHRON, 22 theophylline ext-rel caps 12 hr ; , 22 theophylline ext-rel tabs, 22 theophylline liquid, 22 thiamine, 20 THICK-IT, 19 thioguanine, 7 thyroid, 16 tiagabine, 11 TIAZAC, 10 TIGAN, 16 timolol hemihydrate, 25 timolol maleate, 25 TIMOPTIC, 25 37.

GENERIC BRAND ANTI-INFLAMMATORY AGENTS generic Decadron Fluorometholone generic, FML Forte Forte S.O.P. Prednisolone Acetate generic Econopred Plus Mild Prednisolone Phosphate generic Inflamase Mild BETA-BLOCKERS generic Betoptic S Levobunolol generics only Timolol generics only Timolol Timoptic Ocudose VASOCONSTRICTORS generics only MISCELLANEOUS OPHTHALMIC AGENTS --Cyclosporine Restasis OSTEOPOROSIS AGENTS Alendronate Calcitonin Risedronate Teriparatide OTICS Antipyrine Benzocaine generic AB Otic Glycerin Triethanolamine Cerumenex ANTI-INFECTIVE AND ANTI-INFLAMMATORY COMBINATIONS Acid HC generics only Ofloxacin Floxin Otic Polymyxin-B Neomycin HC generic Cortisporin RESPIRATORY ASTHMA ANTI-ASTHMATIC AGENTS . Montelukast Singulair Zafirlukast Accolate Corticosteroids . Beclomethasone Qvar Budesonide Inhaler Soln Pulmicort Fluticasone Inhaler Rotadisk Flovent Triamcinolone Acetonide Azmacort Sympathomimetics . 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The , i-adrenoceptor agonist isoprenaline potently dilated blood vessels in the posterior capsule of the knee joint, despite its transient lowering effect on systemic blood pressure Fig. 2 ; , which tends to counteract the former effect. These results along with the fact that the dilator responses to adrenaline, isoprenaline and nerve stimulation were virtually abolished by propranolol suggests that , J-adrenoceptors are present in this vascular bed. A previous study from this laboratory using an isolated rabbit knee joint preparation Ferrell & Khoshbaten, 1989 ; failed to demonstrate the presence of f, -adrenoceptors, but such in vitro results have to be interpreted with caution. In a recent in vivo study of the subtypes of a-adrenoceptors in articular blood vessels Najafipour & Ferrell, 1993 ; a2-adrenoceptors predominated, whereas the previous in vitro investigation showed a predominance of aXadrenoceptors Ferrell & Khoshbaten, 1989 ; . Such differences could arise due to the different methodologies used in these studies. Ferrell & Khoshbaten 1989 ; measured changes in perfusion pressure as an indirect indicator of resistance, whereas laser Doppler flowmetry was used in this in vivo study. The former technique is likely to reflect changes in the calibre of the larger resistance vessels, whereas laser Doppler flowmetry reflects changes in the smallest arterioles and in the capillaries. In addition, there may be differences in the responsiveness of blood vessels resulting from changes in vessel tone or the absence of circulating vasoactive substances in vitro. To investigate the nature of these , 8-adrenoceptors, more specific agonists and antagonists were employed. Although the dilator responses to dobutamine, a selective , -agonist Weiner, 1980a ; , were smaller than the responses to isoprenaline Fig. 4A ; , the potent inhibitor effect of the selective , 3-antagonist atenolol Robertson, Kaplan, Caldwel & Speight, 1983 ; on the dilator responses to both of these agents Fig. 4B ; suggests that these effects are mediated via 31-adrenoceptors. The cross-reaction of atenolol with i2adrenoceptors is unlikely because the transient changes in blood pressure due to isoprenaline or salbutamol injection are not significantly different before and after atenolol Fig. SB ; , suggesting that 82-adrenoceptors in other vascular beds are unaffected by atenolol. The difference in potency on blood flow of isoprenaline and dobutamine may reflect differences in their affinities for fl1-adrenoceptors or the additive 2 effect of isoprenaline. The specificity for fi1-adrenoceptors of dobutamine and atenolol is evident from the negligible hypotensive effect of dobutamine, and inability of atenolol to block the depressor effects of isoprenaline and salbutamol Figs 4B and SB ; , which are exerted via systemic ?2-adrenoceptors in other vascular beds, particularly in skeletal muscle. On the other hand, isoprenaline and salbutamol seemed to be equipotent based on their hypotensive effects Fig. SB ; , but the former showed a more powerful vasodilating effect on joint blood vessels Fig. 5 A ; . This may reflect an additive effect on , ll-adrenoceptors. Dobutamine is used in the treatment of cardiac failure mostly for its positive inotropic effect on the human heart. In the present study and at the dose used, it showed no chronotropic effect on the rabbit heart Figs 4A and 6A ; , although isoprenaline showed clear chronotropic effects. This could be due to dobutamine having less chronotropic effects Tuttle & Mills, 1975 ; , or may reflect species differences in , 8-adrenoceptor subtypes in the heart. The 82-agonists salbutamol and terbutaline appeared to be much less potent in joint blood vessels than isoprenaline and dobutamine Figs 4A and SA ; . Although their dilator effects were somewhat decreased by atenolol, they did not differ significantly from control values Fig. SA ; . The , 82-antagonist ICI118551 failed to inhibit the dilator responses to isoprenaline and dobutamine Figs 6B and 7A ; , although the smaller dilator responses to.
I think the risk factors of delivering preemies for outweighs the risks of terbutaline and benazepril.
These drugs function by lowering the amount of stomach acid, which can relieve the pain associated with heartburn, acid indigestion and ulcers. To construct and validate the predictive model, we utilized "claims clues" developed by a bipolar disorder expert consensus panel and tested in the previous casecontrol study. The current study extended the previous findings in additional data sets, using a retrospective cohort design. Data The present study utilized the variables from the development study in 2 different data sets representing 2 common mental health payment scenarios: an allencounters data set and a mental health carve-out data set. The carve-out data set excluded claims from mental health providers; both included all filled prescriptions. Both data sets contained only members of commercially insured nationwide health maintenance organizations or preferred provider organizations aged 18 to 64 years as of January 1, 2001, who were continuously enrolled from January 1, 2000, through September 30, 2003. The all-encounters set contained all claims for 40 244 members who met the age and enrollment criteria. The carve-out set, representing a different health and betahistine.
Sympathetic nervous system stimulants -stimulants ; isoprenaline hydrochloride clenbuterol hydrochloride tulobuterol hydrochloride terbutaline sulphate procaterbol hydrochloride, etc. When it is necessary for a physician to render services in an infusion center, e.g., in the event of an adverse reaction or other medical emergency, the physician may bill for the appropriate evaluation and management service using his or her individual provider number. Documentation should reflect the nature of the emergency and necessity for physician intervention. The medical record must also describe the services rendered by the physician and the time spent in treating the patient. These codes may be used in addition to the E M visit code when there is more than 30 minutes of actual face-to-face physician time required beyond the usual service for the level of the E M code billed. This code should only be used when the physician's expertise is medically necessary in evaluating and managing the patient over a prolonged period and specific documentation describes the content and duration of the service. These codes should only be used in situations requiring constant physician attendance of critically ill or unstable patients for a total of 30 minutes to one hour on a given day. These codes should only be used in situations significantly more complex than other chemotherapy situations and betamethasone. Consecutive days. The daily doses, formoterol 72 g and 120 g, were divided into three administrations over the day in order to mimic a possible clinical situation with patients taking extra inhaled medication for symptom relief. The study was double-blind and randomized and the same number of terbutaline inhalations administered via Turbuhaler was used for comparison. This resulted in daily doses of 6 mg and 10 mg of terbutaline, respectively. The patients used terbutaline on a regular basis during the run-in and washout periods in order to obtain similar baseline values before each treatment period. This treatment, resulted in some degree of tolerance to the systemic 2-receptor mediated effects. It could be assumed, however, that patients prescribed a long-acting 2-agonist also frequently use short-acting 2-agonists. The study design, therefore, also tried to mimic the clinical situation. Theophylline was not a permitted concomitant medication as this was the first high-dose safety study with formoterol Turbuhaler. The safety of the possible combined use of theophylline and high-dose formoterol Turbuhaler needs to be investigated in future studies. The study shows that the same number of inhalations formoterol 6 g or terbutaline 0.5 mg ; from Turbuhaler did not result in equal systemic effects. Six milligrams of terbutaline caused more systemic effects than 72 g formoterol and, similarly, 10 mg terbutaline was more potent than 120 g formoterol. This was true of almost all variables measured. In many countries formoterol is also available as a single capsule-based dry-powder inhaler Aerolizer, Novartis ; . In vitro data indicate a significantly higher pulmonary deposition with Turbuhaler than with Aerolizer Astra Draco, data on file ; . It is therefore likely that the clinical efficacy of 6 g formoterol Turbuhaler corresponds to that of 12 g formoterol Aerolizer. Patients used to taking a defined number of inhalations for acute symptom relief from one inhaler will probably also take the same number of doses from another inhaler. From that point of view, it is valuable to know that the same number of inhalations via Turbuhaler of formoterol gave less systemic activity than the same number of inhalations of terbutaline. However, the exact potency relationship between formoterol and terbufaline could not be estimated because of the study design used, i.e. two parts with different patient populations and only one dose of each drug in each part. The largest differences in systemic effects between formoterol and terbjtaline were obtained for serum potassium values, with 10 mg terbutalune causing greater potassiumlowering effects than 120 g formoterol. Although all patients included were asthma patients on regular treatment with 2-agonists, there was a gradual increase in potassium values over the 3 study days, probably indicating a further development of tolerance to the extrapulmonary effects. However, as no placebo group was included, this conclusion cannot be drawn with certainty. An interesting finding in this study was that the systemic effects of formoterol had the same time profile as those of terbutaline. This indicates that formoterol is a short-acting 2-agonist in all respects except for the long bronchodilatation seen after inhalation. This is also in agreement with the findings in another study where the rapid normalization of formoterol-induced hypokalaemia after a cumulative dose of 18 + min intervals ; followed the same time course as after cumulative.

Figure 4 Change in [35S]GTPgS binding to rat uterine membrane from days 15, 18, 20 and 22 of pregnancy by various concentrations of terbutaline. Points are means S.E.M. from 3 separate experiments carried out in triplicate. The basal value is that of [35S]GTPgS binding without terbutaline stimulation and was regarded as 0 and bethanechol.
Order bricanyl online bricanyl price comparison bricanyl - order online no prescription required prior to ordering home medications by brand name aceon bricanyl price comparison - order bricanyl online bricanyl information bricanyl terbutaline sulphate ; is used to prevent and treat wheezing, shortness of breath, and troubled breathing caused by asthma, chronic bronchitis, emphysema, and other lung diseases.

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NO. 1 2 3 DIALOGWEB FORMAT --Medium Long Free -Short Full -RECORD CONTENT DIALOG Accession Number Bibliographic Data including Controlled Substance Section ; and Category Section Bibliographic Data including Controlled Substance Section ; Bibliographic Data including Controlled Substance Section ; , Category Section, and Pharmacology Section Title and Word Count Full Record Title, Word Count, and VA Classification Full Record KWIC Key Word In Context ; displays a window of text; may be used alone or with other formats For an explanation, enter HELP TYPE, HELP UDF, HELP TAG online. TYPE S3 TI, CC 1-5 TYPE S3 5 1-10 TAG TYPE 00001091 5 DISPLAY 00000832 TI, CC PRINT 00000964 9 and urecholine.

Incomplete communication and documentation Selected Case Reports Case #1: An IV pump was originally programmed to deliver 2 mL minute of a pitocin infusion but was inadvertently changed to deliver 20 mL minute. This resulted in the patient receiving a 10-fold overdose for several minutes before the error was discovered. The patient developed tetanic contractions lasting 6 minutes. The pitocin IV was stopped, the patient's obstetrician was called and the patient was given a bolus of fluid, terbutaline, and oxygen. The baby's fetal heart rate experienced 5 minutes of deceleration in the 50 - 80 beats per minute range, but returned to baseline in the120's. The uterine tone also returned to normal. Case #2: An epidural infusion consisting of bupivacaine 0.1% with epinephrine and 0.8 mcg mL fentanyl was ordered to infuse at 15mL hour. The patient was also receiving lactated ringers solution as the maintenance IV to be infused at 125mL hour. A nurse discovered however, that the epidural IV line in channel A of the IV pump ; was infusing at 125mL hour while the lactated ringers was labeled as an epidural in channel B infusing at 15mL hr. The patient inadvertently received the excessive dose of bupivacaine epinephrine fentanyl over approximately 2 hours. The infusions were stopped, ephedrine and fluids were given bolus, vitals monitored every 5 minutes and the epidural was resumed when the patient was ready for delivery. There were no adverse outcomes for the mother or baby. Case #3: In preparing for a scheduled caesarian section, the anesthesia provider prepared a pitocin infusion 20 units in 1000 mL ; that was intended to be given following delivery of the placenta. The IV tubing was primed with the pitocin solution, however, the anesthesia provider failed to close the roller clamp. About 300 mL of the pitocin solution rapidly infused prior to the induction of anesthesia or the surgical incision. The patient experienced titanic uterine contractions and the baby's heart rate decreased to 60-70 beats per minute. An emergency caesarian section had to be performed. The mother and baby recovered without permanent harm. Case #4: Upon admission, a patient stated she was allergic to sulfa and cefaclor. A physician in labor and delivery ordered cefotetan 1 gram IV now. After the drug was administered, the patient developed a flushed face, welts, and complained of itching on the face, back, and chest. Case #5: A physician ordered insulin 10 units in 100mL of normal saline to be infused at 1 unit hour 10mL hour or 0.1unit mL ; . The pharmacy prepared the IV bag as ordered, however, the administering nurse thought the standard insulin concentration was 1unit mL and therefore programmed the pump to infuse the insulin bag at 1 mL hour 0.1unit hour ; resulting in a 10 fold under dosing. Several hours later, the patient's blood glucose levels were elevated prompting the physician to order an increase in the insulin infusion rate to 11 mL hour. The error was discovered when nursing requested an additional IV bag from the pharmacy.

FIG. 3. Combined effects of terbutaline, TPA, and OAG on phosphatidylcholine secretion. The alveolar type I1 cells, which and bicalutamide.

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As of June 2002: 80 countries had expressed interest, 39 countries had developed plans of action, 22 countries had entered negotiations with pharmaceutical companies, and 19 had successful UN-brokered supply agreements for ARVs. From July 2002, two regional groups have formed to negotiate with pharmaceutical companies with WHO UNAIDS support: ECOWAS 15 West African countries ; and CARICOM 15 Caribbean countries.

In one study 4 ; of patients with brittle asthma defined by wide diurnal variations in pef rates ; , twice-daily subcutaneous administration of terbutaline improved symptoms, medication use, and pef rates and casodex and terbutaline. T TAGAMET .35 TALACEN .15 TALADINE.35 TALWIN NX.16 TAMBOCOR.19 TAMIFLU .5 tamoxifen citrate.10 tana pse.46 tana r-12 .46 tanacof-a 12 .46 TANAFED.47 TANAFED DP.47 tanatan rf .46 tanavan.46 TAPAZOLE .31 TARABINE PFS.11 TARCEVA.11 TARGRETIN.11 TAVIST.44 TAXOL .12 TAXOTERE .12 TAZICEF.6 TAZORAC.25 taztia XT .20 tbc .27 TE ANATOXAL BERNA .36 tebamide.33 TEGRETOL.12 TEGRETOL XR .12 TEMOVATE .27 TEMOVATE EMOLLIENT .27 TENEX .19 TENORETIC 100.21 TENORETIC 50 .21 TENORMIN .20 TENORMIN I.V 20 terak.40 TERAZOL.38 TERAZOL 7 .38 terazosin .48 terazosin HCl .19 terbutaline sulfate.47 terconazole.38 TERRAMYCIN IM.9 TERRAMYCIN W POLYMYXIN .40 tesamone-100.32 TESLAC .12 TESTIM .32 testomar .28 testosterone.32 testosterone cypionate .32 testosterone enanthate .32 testosterone propionate .32 75. The allele distribution of the AHSG polymorphism Thr230Met was in Hardy-Weinberg equilibrium, and the frequency of the minor allele T Met ; was 40%. In Table 1, the effect of Thr230Met on clinical parameters is shown. No effect of the genotype on any of the examined clinical parameters was found. The results of the subcutaneous fat cell lipolysis measurements are shown in Table 2. There were no differences between the genotypes, either in basal lipolysis, in maximum stimulation of lipolysis induced by dobutamine, terbutaline, or forskolin or in maximum inhibition of lipolysis induced by clonidine. In contrast, with respect to lipolytic adrenoceptor sensitivity, a marked effect was found for terbutaline. Men homozygous for the AHSG rs4917 Met allele had approximately 1.5 log units higher sensitivity for adipocyte 2-adrenoceptor stimulation than men heterozygous or homozygous for the Thr allele P 0.0008 comparing three genotypes, P 0.0024 after Bonferroni adjustment for multiple comparison of adrenoceptor subtype sensitivity ; . In other words, 2-adrenoceptor sensitivity of fat cells from Met-allele-homozygous men was about 35-fold higher than that of fat cells from men with Thr Met or Thr Thr genotype Fig. 1 ; . The genotype effect on terbutaline sensitivity remained statistically and bisoprolol. Placebo in trials to assess the efficacy of new antidepressant medications.11 It has been argued that the use of placebo assignment in such trials remains justified because the demonstraAuthor Affiliations and Financial Disclosures are listed at the end of this article. Corresponding Author and Reprints: B. Timothy Walsh, MD, Department of Psychiatry, College of Physicians and Surgeons of Columbia University, 1051 Riverside Dr, Unit 98, New York, NY 10032. SYMAX-SR SYMBYAX SYMLIN SYMMETREL SYNAGIS SYNALAR SYNALGOS-DC SYNAREL SYNERA SYNERCID SYNTHROID SYPRINE syringe w-ndl, disp., insulin T-4 GEL TABLOID TACLONEX TAGAMET TALACEN TALADINE TALWIN TALWIN NX TAMBOCOR TAMIFLU CAPSULES TAMIFLU SUSPENSION tamoxifen citrate TANA PSE TANA R-12 TANACOF-XR TANAFED DP TANATAN RF TANAVAN TANNATE PEDIATRIC TAPAZOLE TARCEVA TARGRETIN CAPSULES TARGRETIN GEL TARKA TASMAR 119 47 55 TAXOTERE TAZICEF TAZORAC TAZTIA XT TE ANATOXAL BERNA TEGRETOL TEGRETOL-XR TEMOVATE TEMOVATE E TENEX TENORETIC 100 TENORMIN TERAZOL 3 TERAZOL 7 terazosin hcl terbutaline sulfate terconazole TERNAMAR TESLAC TESTIM TESTOPEL testosterone testosterone cypionate testosterone enanthate testosterone propionate TESTRED tetanus toxoid TETANUS TOXOID ADSORBED TETANUS DIPHTHERIA TOXOID tetracycline hydrochloride TETRA-MAG TEVETEN 400MG TEVETEN 600MG TEVETEN HCT TEV-TROPIN TEXACORT THALITONE THALOMID 37 33 64 THEO-24 THEOCAP THEOCHRON THEOMAR GG theophylline theophylline cr theophylline er theophylline sr theophylline td THERACYS THERA-FLUR-N THERMAZENE THIOLA thioridazine hydrochloride thiotepa thiothixene THYMOGLOBULIN thyroid THYROLAR-1 TIAZAC 120MG TIAZAC 180MG TIAZAC 240, 300, 360, TICE BCG TICLID ticlopidine hydrochloride TIGAN TIKOSYN TILADE TIME-HIST TIMENTIN TIMOLIDE timolol maleate timolol maleate ophthalmic TIMOPTIC TIMOPTIC OCUDOSE TIMOPTIC-XE TINDAMAX TIS-U-SOL 24 tizanidine hydrochloride T-NAF TOBI TOBRADEX tobramycin sulfate tobramycin sulfate and sodium chloride TOBRASOL TOBREX TOFRANIL TOFRANIL-PM tolazamide tolbutamide TOLECTIN DS TOLINASE tolmetin sodium TOPAMAX TOPICORT TOPICORT LP TOPOSAR TOPROL XL TORADOL ORAL torsemide TOURO ALLERGY TOURO LA TOURO LA-LD T-PERIO TPN ELECTROLYTES FTV TRAC TRACLEER tramadol hcl tramadol hcl and acetaminophen TRANDATE TRANDATE IV TRANSDERM-SCOP tranylcypromine sulfate TRAVASOL TRAVATAN TRAVATAN Z. Cairns & dulhunty 1993b ; showed that terbutaline's force potentiating effects on rat soleus muscle fibres were not due to changes in the action potential, na + k + pump activity, extracellular ca 2 + influx, or glycolysis, and speculated that the drug acted through a cyclic amp-dependent increase in sr ca release. Table 23.1. Additional Dicarboxylate-containing Angiotensin Converting Enzyme Inhibitors, because terbutaline pharmacology.
39. Siimes ASI, Creasy RK. Effect of ritodrine on uterine activity, heart rate, and blood pressure in the pregnant sheep: combined use of alpha or beta blockade. J Obstet Gynecol 1976; 126: 1003-1010. Barden TP, Peter JB, Merkats IR. Ritodrine hydrochloride: a betamimetic agent for use in preterm labor. I. Pharmacology, clinical history, administration, side effects, and safety. Obstet Gynecol 1980; 56: 1-6. Merkatz IR, Peter JB, Barden TP Ritodrine hydrochloride: a betamemetic agent for use in preterm labor. II. Evidence of efficacy. Obstet Gynecol 1980; 56: 7-12. Beall MH, Edgar BW, Paul RH, Smith-Wallace T. A comparison of ritodrine, terbutaline, and magnesium sulfate for the suppression of preterm labor. J Obstet Gynecol 1985; 153: 854-859 and baclofen.
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The chromatographic separations of the enantiomers of a series of eight phenylhydantoin and methylhydantoin derivatives of amino acids are described, using reversed-phase HPLC with a chiral additive in the mobile phase: native 3-cycodextrin 3-CD ; , carboxymethylated fl-CD and a cationic fl-CD derivative. Retentions time and selectivities obtained with these three chiral additives are compared. As the cationic 3-CD gave promising results, its usefulness in the resolution of several drugs of particular interest chlorthalidone, terbutaline, mephobarbital and hexobarbital ; is demonstrated.
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